John D. Barrow, renowned Cambridge cosmologist and author, director of the Millennium Mathematics Project, and regular contributor to Plus has won the Royal Society Michael Faraday Prize, which is awarded annually for excellence in communicating science to the general public. The prize
lecture, entitled Every picture tells a story, took place in London last week and is a fascinating tour through scientific imagery. It has now been put online on the Royal Society website. Happy watching!
To hear a version of Barrow's talk focusing on mathematics, listen to the enhanced Plus podcast Cosmic imagery. It comes with pictures!
We may be in a recession, but according to Gordon Brown, that's all the more reason to invest in science and maths education. In a speech in Oxford today, Brown pledged to increase the number of students taking maths A level from the current 56,000 to 86,000 in 2014, and to make sure that 90% of state schools teach physics, chemistry and biology as separate subjects, also by 2014. The measures
are meant to help ensure Britain's future competitiveness, and ease its over-dependence on financial services, which have got us into this mess in the first place. Some of the losers of the recession, those who have lost their jobs but have maths, science and IT degrees, are to be encouraged and supported to take up teaching, to tackle the lack of specialist teachers in these subjects. Brown also
pledged to encourage a positive public debate about science in order to improve the public's understanding of its role in society.
Geneticists are usually concerned with picking apart the individual genes that make up a genome, but now two biochemical engineers from the University of Wisconsin Madison have decided to re-assemble all the pieces and give them a good shake. They found that it's not just the genes themselves, but also the way in which they are organised within the genome, that determine the characteristics of
This type of permutation is exactly what keeps us "interesting" despite greater evidence of mechanistic implementation and structure in biology. Even given the fact that "not all permutations are equally likely" the variation in time and space and the Hilbert spaces of DNA, neural nets and life threads are still enormously re-combinatorial.
It's not often that maths makes it into the mainstream media, and when it does, it's usually a very specific bit of maths — a statistical result or a certain application — that's being examined. An article published yesterday on the Guardian website makes a nice exception. It explores
how maths underlies almost every aspect of modern life, from traffic lights to iTunes. Thank you, Guardian!
You can find out more about some of the topics mentioned in the article on Plus:
Sometimes a boring story can become a lot more interesting if you do some skilled number juggling. This is what seems to have happened in an article in The Daily Telegraph, which claims that 90% of us carry a gene which increases the risk of high
blood pressure by 18%. And high blood pressure is of course linked to dreaded killers like stroke and heart disease.
In his Understanding Uncertainty blog David Spiegelhalter traces the story back to a paper in Nature Genetics. The authors of the paper investigate three gene variants that can occur in the human genome. Two of them are rare, only about 10% of the population
carry them, while the third is present in 90% of the population. The authors show that the gene variants are associated to proteins called natriuretic peptides, which are linked to blood pressure (this is the main point of the paper, since such a genetic connection had never before been found). The two rarer gene variants, according to the paper, reduce the risk of high blood pressure by
15%. The phrase "18% increased risk of high blood pressure" appears nowhere in the paper. Rather it's the result of some creative accounting on the part of someone operating in the media chain which links the actual paper to the final article in The Daily Telegraph. Here's how it's done:
Say that the risk of high blood pressure for someone carrying the more common gene variant is x. Now the 15% decrease associated to the two less common variants takes this down to 0.85x. To get back to x, we need to add 0.15x, and this is exactly 17.647% of 0.85x — hence the claim of an 18% increase in risk.
The calculation is undoubtedly correct, but it puts a spin on the story. Rather than taking the common case as a base line and talking about the risk reduction associated to the less common cases, it does things the other way around. This is in stark contrast to the paper's authors own turn of phrase, which links the rarer variants to risk reduction, but says that the common variant "was not
significantly associated with either systolic or diastolic blood pressure." It's a bit like noting that some people live to 110 and then complaining that most of us die prematurely. "This is a masterful piece of re-framing of the evidence," says Spiegelhalter on his blog. "Not exactly wrong, but definitely changing the story. Just like a
change from 98% to 96% in a survival rate seems a lot more innocuous than a doubling of the mortality rate from 2% to 4%."